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Tag «blood sugar watch for diabetics»

Session 12 – Diabetic Health Clinic

In our last newsletter we mentioned Subway and the fact that we are going to have a look at a subway meal. when I started I was over 90 kg and on 3 medications and still my blood sugars were up to 13.5 on waking. This sometimes is not possible because of a lack of organic produce or because it may be price prohibitive. I started the DHC program because I was very unhappy with myself, very over weight and couldn’t see how I was ever going to lose the weight, it seemed impossible as I had tried everything you could imagine. The main ingredients are zucchinis and walnuts so there are some very important health benefits. The programme was real eye opener. What we are surprised about is the fact that in regulated markets where governments are regulating health destroying industries, that the food industry avoids scrutiny and regulation.

I will be emailing you after tomorrows call with instructions on how to use it. What if we defined health. I became a diabetic even though I lived a

Full Text Article: Differences in pulsatile ocular blood flow among three classifications of diabetic retinopathy.

The efficiency of two non-stereo photographs of the posterior pole as a screening device in long-term follow-up for the detection of proliferative diabetic retinopathy was evaluated in a clinical setting among 1341 diabetic hospital patients. Although panretinal photocoagulation (PRP) is the treatment of choice for PDR, it has secondary effects that can affect vision. The incidence of diabetic retinopathy has increased with the increase in the long-term survival of diabetics (Richter, 1987). Diabetic retinopathy occurs in more than half of the people who develop diabetes. These include panretinal photocoagulation (PRP), using the argon laser or xenon arc, for proliferative retinopathy, and focal photocoagulation for macular oedema. Proliferative retinopathy, with cataract and macular edema, was identified in both eyes. After a mean follow up of 13 months all eyes in the krypton group showed a reduction or complete regression of PDR; in the retreated group 10 eyes improved and two

The diabetic antigen glutamic acid decarboxylase (GAD 65) in the human peripheral blood. – PubMed

We evaluated the frequency of antibodies to glutamic acid decarboxylase (GAD-Ab) in Japanese patients diagnosed initially as having non-insulin-dependent diabetes mellitus (NIDDM) and investigated a possible link between the presence of GAD-Ab and development of the insulin-dependent (ID) state. Sera were collected from 40 NIDDM patients, who had history of secondary failure to treatment with sulfonylurea, for analysis of fasting c-peptide and GAD-Ab. Birth data and information on parental age and diabetes were obtained from Danish registers. In the vaccinated animals, antibody responses to GAD65 or to GAD67 were induced. Univariate associations with insulin treatment < or = 1 year from diagnosis included GADab positivity, no family history of diabetes, lower BMI for men, and GADab positivity and lower BMI for women. Of the 38 sera positive for anti-GAD by RIP, only 6 had antibodies detectable by Western blotting, and all gave an RIP titer of at least 1:250. One single immunoglobulin apheresis decreased IgG by 66.2 +/- 9.1%, IgA by 66.8 +/- 8.7%, and IgM by 57.7 +/- 12.9%. In China, patients initially diagnosed as NIDDM may in many cases suffer from LADA. On repeated testing 18 months later, her anti-glutamic acid decarboxylase antibody levels had declined by more than 85%. Given that Th2 effector cells can cause pathology, the glutamic acid decarboxylase-bifunctional peptide inhibitor may represent a novel mechanism to induce interleukin-4 without Th2-associated pathology. Our experimental results may also be important for the therapy of diabetes mellitus type 1 and other autoimmune diseases by the passive administration of GAD 65 antibodies.